Information processing in sleep-onset insomnia

Information processing in sleep-onset insomnia by Rona S. Kertesz Download PDF EPUB FB2

The present thesis study is a systematic investigation of information processing at sleep onset, using auditory event-related potentials (ERPs) as a test of the neurocognitive model of insomnia.

Insomnia is an extremely prevalent disorder in society resulting in problems with daytime functioning (e.g., memory, concentration, job performance Author: Rona S. Kertesz. Towards an affective information-processing theory of sleep onset and insomnia. (Paper to be presented at ISRE).

We develop a cognitive-affective theory of sleep onset and insomnia. We develop a cognitive-affective theory of sleep onset and insomnia (Beaudoin,). This somnolent information processing theory is Information processing in sleep-onset insomnia book (Artificial Intelligence-inspired).

We argue that the two-process model of sleep (Borbély,) is necessary, but insufficient because it ignores pro-somnolent and insomnolent factors Author: Luc P. Beaudoin and Sylwia Hyniewska. This paper illustrates how from information processing theory, psychotherapy and self-help applications can be derived and targeted at a problem that affects a very large number of people (insomnia).

The theory in question is broad in that it involves cognitive, affective and executive mechanisms across a proposed mental architectureAuthor: Luc P. Beaudoin. mentation. Harvey et al. also point to mentally processing active concerns as insomnolent.

The construct of concern, being theoretically critical, needs a theoretical framework.5 More generally, to align sleep research with cognitive science, one needs an information-processing framework that characterizes the processing of goals, other.

Event-related potentials may be applied to directly measure information-processing Information processing in sleep-onset insomnia book associated with the problem of insomnia. This study is a systematic investigation of cortical hyperarousal during the sleep-onset period in participants with sleep-onset insomnia complaints.

Thirteen poor sleepers and twelve good sleepers (GS) were administered an oddball task while awake in the morning.

Theoretical ERP waveforms following standard and deviant stimuli in good sleepers (G5) and patients with primary insomnia (PI) in an alert waking state (1A and 2A) and at sleep onset (16 and 26).

Jennie M. Kuckertz, Nader Amir, in Social Anxiety (Third Edition), Introduction. Psychopathologists have been greatly influenced by information-processing models of emotional disorders that suggest preferential processing of threat-relevant information (i.e., information-processing biases) underlie these disorders (Beck, Emery & Greenberg, ; Williams Watts, MacLeod, & Mathews, This is the text book sleep onset insomnia definition, an inability to fall asleep despite your physical tiredness.

These issues can be chronic and ongoing, or acute and short term. And the symptoms of sleep onset insomnia will vary depending on how it presents for you. It’s important to understand what your body is saying when this happens.

INTRODUCTION. Insomnia is the most prevalent sleep disorder and is associated with increased risk of psychiatric and cardiometabolic morbidity. 1,2 Data from multiple systems, such as cognitive-emotional, stress, cardiovascular, metabolic, and immune, support that insomnia is a disorder of h hyperarousal.

3 –5 Insomnia in adults has been associated with anxious-ruminative traits. Efficacy study: In an experimental model of transient insomnia, exposure to the Nightingale smart home sleep system significantly reduced sleep onset latency by 38% compared to normal environmental noise in a group of healthy subjects.

This study was conducted between June and completed in February   Information Processing at Sleep Onset and During Sleep in Patients With Insomnia (COTE) The safety and scientific validity of this study is the responsibility of the study sponsor and investigators.

Listing a study does not mean it has been evaluated by the U.S. Federal Government. I should like to offer two brief comments on the interesting article on psychophysiological insomnia by Perlis et al. In this article, which proposes several useful and testable hypotheses, the authors suggest that one possible explanation for the discrepancy between subjective reports of being awake while the EEG shows unequivocal evidence of sleep is that the patient with.

Sleep onset marks the transition from a fully awake and conscious state to one of sleep and unconsciousness. The sleep onset period thus provides an elegant means to study the sudden change from a state of conscious to unconsciousness, the change from a state in which information is actively processed to one in which it is now inhibited.

This volume investigates recent work on implicit memory and other aspects of information processing outside of awareness. It explores the role of the sleep laboratory as a vehicle for studying various aspects of information processing outside of awareness.

Trait anxiety and sleep-onset insomnia: Evaluation of treatment using anxiety management training. Journal of Psychosomatic Research, 54(1), Retrieved from Science Direct Database. Lichstein, K. L.(Aug. Behavioral. Types of Insomnia. Many people may associate insomnia with the inability to fall asleep but the sleep disorder is much more complicated than that.

Different types of insomnia exist: Sleep onset insomnia is a sleep problem that involves the inability to fall asleep within a reasonable amount of time. According to the National Sleep Foundation. Psychophysiological insomnia, as defined in the ICSD-2, is a state of “heightened arousal and learned sleep preventing associations that result in a complaint of insomnia and associated decreased functioning during wakefulness.”The specific criteria are as follows (page 7): • Complaint of difficulty in initiating sleep or in maintaining sleep, waking up too early, or sleep that is.

The guideline represents the first comprehensive, evidence-based analysis of medications commonly used to treat people experiencing persistent trouble falling asleep and staying asleep, Michael J.

Sateia, M.D., chair of the guideline development task force, told Psychiatric News. Medications referenced in the guideline include sedative-hypnotics, antidepressants, and other prescription drugs. Sleep is a naturally recurring state of mind and body, characterized by altered consciousness, relatively inhibited sensory activity, reduced muscle activity and inhibition of nearly all voluntary muscles during rapid eye movement (REM) sleep, and reduced interactions with surroundings.

It is distinguished from wakefulness by a decreased ability to react to stimuli, but more reactive than a. Event-related potentials may be applied to directly measure information-processing deficits associated with the problem of insomnia.

This study is a systematic investigation of cortical hyperarousal during the sleep-onset period in participants with sleep-onset insomnia complaints. Handbook of Sleep Research, Vol provides a comprehensive review of the current status of the neuroscience of sleep begins with an overview of the neural, hormonal and genetic mechanisms of sleep and wake regulation before outlining the various proposed functions of sleep and the role it plays in plasticity, and in learning and memory.

Typically, the first REM episode occurs about minutes after sleep onset. In depressed persons, however, it often appears much earlier, often minutes after falling asleep.

This has led some researchers to believe that there are abnormalities of the body’s rhythms which. (Primary) Insomnia Insomnia is a sleeping disorder characterised by (DSM-IV-TR, ): ¾Sleep-onset or sleep maintenance complaints, and/or decreased sleep quality, presenting itself for at least one month ¾Significant impairment in daytime functioning ¾No other psychological, psychiatric or medical problems that can cause the insomnia complaint.

Patients with chronic insomnia, prevalent in 10–15% of the adult population, who underwent the subjective experience of chronically disturbed sleep, sleep loss, non-refreshing sleep, and heightened arousal in bed with impaired quality of life, showed a decreased ability to disengage from external information processing at sleep on-set ().They frequently experience intrusive thoughts.

Recently, Kay and colleagues reported that patients with insomnia who exhibit greater subjective–objective sleep discrepancy—discrepancy between sleep diary reports and polysomnography (PSG) sleep onset latency—differed with respect to glucose metabolism in the insula and the left anterior cingulate cortex (ACC) during NREM sleep.

This. The Overcoming Insomnia Course will improve your health, optimize your brain, and teach you evidence-based sleep strategies to maintain better sleep hygiene for the rest of your life.

WHAT YOU GET: 21 exclusive evidence-based lessons ($1, Value). Melatonin for chronic sleep onset insomnia in children: a randomized placebo-controlled trial.  J Child Neurol. ;16(2) doi: / PubMed Google Scholar Crossref. The extent of continuance of melatonin therapy initiated in pre-pubertal children with chronic sleep onset insomnia (CSOI) was investigated in young adult life.

Sleep timing, sleep quality, adverse events, reasons for cessation of therapy, and patient characteristics with regard to therapy regimen, chronotype and lifestyle factors possibly influencing sleeping behavior were assessed. Insomnia disorder is characterized by chronic dissatisfaction with sleep quantity or quality that is associated with difficulty falling asleep, frequent nighttime awakenings with difficulty returning to sleep, and/or awakening earlier in the morning than desired.

Although progress has been made in our understanding of the nature, etiology, and pathophysiology of insomnia, there is still no. INSOMNIA IS ESTIMATED TO AFFECT APPROXIMATELY 10% OF THE ADULT POPULATION 1 AND IS CHARACTERIZED BY DIFFICULTIES WITH INITIATING AND/ or maintaining sleep, or non-restorative sleep, accompanied by daytime impairment.

2 Primary insomnia (PI), a largely exclusionary diagnosis of poor sleep, ruling out psychiatric, medical and additional sleep-related. Information-processing and experience that the brain tends uniquely to produce during sleep-onset is pro-somnolent.

That means that the sleep onset control system contains a positive feedback loop. If this is true, then other things being equal, if you can engage in mentation that is both counter-insomnolent and pro-somnolent, you are more. INTRODUCTION. Chronic primary insomnia, also termed psychophysiological insomnia, is a disorder characterized by chronically disturbed sleep and sleep loss, nonrefreshing sleep, and heightened arousal in bed; these symptoms have a profound effect on psychological well-being, as well as on social and cognitive functioning.

1 Patients with chronic primary insomnia commonly have daytime cognitive. Despite complaints of difficulties in waking socioemotional functioning by individuals with insomnia, only a few studies have investigated emotion processing performance in this group.

Additionally, the role of sleep in socioemotional processing has not been investigated extensively nor using quantitative measures of sleep. Individuals with insomnia symptoms (n = 14) and healthy good sleepers.

R21MH Information processing at sleep onset and during sleep in patients with insomnia. Kimberly Cote, PhD is a Professor of Psychology and Neuroscience at Brock University in St. Catharine’s, Ontario, Canada. She supervises MA and PhD students in the behavioral neuroscience stream of the graduate program in Psychology.

Event-Related Potentials During the Transition to Sleep for Individuals With Sleep-Onset Insomnia. Behavioral Sleep Medicine: Vol. 9, No. 2, pp. Cognitive behavioral therapy for insomnia (CBT-I or CBTI) is a short, structured, and evidence-based approach to combating the frustrating symptoms of insomnia.

How Does CBT-I Work. CBT-I focuses on exploring the connection between the way we think, the things we do, and how we sleep. The neuroscience of sleep is the study of the neuroscientific and physiological basis of the nature of sleep and its functions.

Traditionally, sleep has been studied as part of psychology and medicine. The study of sleep from a neuroscience perspective grew to prominence with advances in technology and proliferation of neuroscience research from the second half of the twentieth century.

Chronic insomnia is a common sleep problem and there is a need to complement the existing treatment options. Yoga nidra practice is documented to be used for sleep by sages.

Recently, yoga nidra has been used in patients of menstrual abnormalities, post- traumatic stress disorder, diabetes, anxiety and depression but little is known about its effect on sleep or sleep disorders. My main scholarly interests are in adult self-regulated learning, expertise, sleep onset and insomnia.

I specify and approach psychological research problems in terms of the global information-processing architecture and interacting mechanisms (motivation, emotions, moods, executive functions, more typically cognitive functions) of mind. Processing speed implies a greater ability to easily do simple or previously-learned tasks.

This refers to the ability to automatically process information, which means processing information quickly and without doing it consciously.

The higher the processing speed, the more efficient you are able to. In patients with paradoxical insomnia, also called sleep state misperception, there is a significant discrepancy between objective sleep quality and subjective perception of sleep.Insomnia affects approximately 10 to 17 percent of the adult U.S.

population.1, 2 In contrast to the occasional sleepless night experienced by most people, insomnia may be a persistent or.